Tea Tree Oil Usage

1. Anti-bacterial, Antiseptic, Antioxidant

Tea tree oil has long been used to get rid of the bacteria that cause body odor and acne. Recent research indicates it is also effective against some fairly deadly bacteria such as methicillin-resistant staphylococcus aureus, better known as MRSA, which is currently plaguing our hospitals and medical facilities.

American researchers reported in 1999 that one of the ways tea tree oil helps is by activating white blood cells which are part of the immune system. French researchers investigating tea tree oil in 2006 stated, “Melaleuca alternifolia essential oil may not only act as an anti-inflammatory mediator through its antioxidant activity but may also efficiently protect the organism by reducing the proliferation of inflammatory cells without affecting their capacity to secrete anti-inflammatory cytokines.”

October 2016 research appearing in Applied Microbiology and Biotechnology by Chinese researchers found that tea tree oil was quite effective against Escherichia coli, Staphylococcus aureus (golden staph), and Candida albicans. It worked by compromising the cell membrane of these bacteria and fungi, ultimately leading to cell death.

One of the problems associated with widespread use of antibiotics is that bacteria are developing resistance to these drugs. In 2015, Brazilian researchers found that tea tree oil was effective against Staphylococcus aureus which exhibits resistance to multiple antibiotic drugs.

Interestingly, Malaysian researchers published an article in 2013 in the journal Phytomedicine which investigated the relationship between various essential oils and antibiotics. The researchers found that none of the oils evaluated (including melaleuca alternifolia) caused an adverse reaction when taken with various antibiotic drugs. They found that the use of some of the essential oils (namely peppermint, cinnamon, and lavender) along with antibiotics provided a synergistic effect!

2. Anti-cancer

Joint Australian and American research reported in Journal of Dermatological Science in 2012 found that tumor-bearing mice given a combination of melaleuca alternifolia and dimethyl sulphoxide (DMSO) activated an immune response and resulted in tumor cell death.

Two 2010 Australian studies with mice found that tea tree oil and its major active terpene component, terpinen-4-ol, had anti-tumor activity against two cancer cell lines: mesothelioma and melanoma. Researchers stated that tea tree oil “significantly inhibited the growth” of these two tumor cell types.

Chinese researchers in 2009 reported that tea tree oil had both antimicrobial and anti-cancer effects. They found that tea tree oil exhibited strong cytotoxicity (toxic to cancer cells) against human lung cancer cells, human breast cancer cells (MCF-7, which are hormone receptor positive, infiltrating ductal carcinoma) and human prostate cancer cells (PC-3, which exhibit high potential for metastasis).

Italian research reported in 2004 investigated tea tree oil against human melanoma (M14 WT) cells, as well as Adriamycin-resistant melanoma cells. Tea tree oil induced apoptosis (planned cell death, lacking in cancer cells) against both cell lines but especially the Adriamycin-resistant cells.

3. Anti-fungal, Anti-mold

Tea tree oil has been used for decades to help treat conditions like jock itch, nail fungus, and athlete’s foot because of its excellent anti-fungal properties.

In 2016 Italian researchers scrutinized several different essential oils including basil, mint, tea tree, lavender, winter savory, and oregano for their antifungal effects against Candida albicans. They discovered the essential oils inhibited both the growth and activity of Candida albicans more efficiently than a popular antifungal drug, clotrimazole.

A 2015 Australian study investigating fungal contamination in indoor environments looked at tea tree oil and four other agents, including industrial strength disinfectants. Researchers noted that of the five substances tested, the tea tree oil exhibited the best inhibitory effect on the growth of the two fungi studied. Tea tree oil was just as effective applied as a liquid or vapor.

Cancer patients often suffer from oral yeast infections due to the treatments they undergo. In 2006, UK researchers found that tea tree oil was effective against 301 different kinds of fungal infections found in the mouths of 199 patients with advanced cancer. Researchers stated that the tea tree oil was even effective against 41 fungi that were known to be resistant to traditional antifungal drugs.

Older research reported in 1992 by Australian researchers found that tea tree oil was quite effective in treating the symptoms of tinea pedia (athlete’s foot). They noted that tea tree oil was just as effective as tolnaftate, a popular anti-fungal agent.

4. Anti-inflammatory, Immune Stimulating

A plethora of research studies exist attesting to the anti-inflammatory properties of tea tree oil. In 2014, Brazilian researchers found that in addition to being an anti-inflammatory, tea tree oil also stimulated human macrophages, a type of white blood cell of the immune system involved in engulfing and destroying bacteria, cancer cells, microbes, etc.

5. Antiparasitic

A study reported in May 2016 by Brazilian researchers discovered that tea tree oil was effective against the eggs and larvae of Haemonchus contortus (aka the barber’s pole worm, a common parasite of ruminants).

A 2009 Italian study found that tea tree oil was effective against equine ringworm.

6. Anti-viral

Tea tree oil has long been used for easing the pain and itching of chickenpox and cold sores, both caused by the herpes simplex virus. Tea tree has also been found to be helpful against influenza; even the H1N1 virus strain.

7. Respiratory Complaints

Tea tree oil has a lengthy history of use for easing lung congestion and infections. Indeed, a Chinese animal study reported in 2016 found that a nanoemulsion of tea tree oil was effective against fungal and bacterial pneumonia due to its anti-microbial, anti-fungal, and anti-inflammatory properties.

Tea tree oil is also very effective against the bacterium that causes Legionnaire’s Disease, which adversely affects the lungs much like pneumonia. Italian scientists reported in 2009 that Legionella pneumophila was “exquisitely sensitive” to tea tree oil. They further stated that tea tree oil should be investigated for use as an anti-Legionella disinfectant, and for control of water system contamination in places like spas, small water lines, and in respiratory medical devices.

8. Insect Repellent

Tea tree oil has been used by indigenous Australians for hundreds of years as an insect repellent, and also as an aid to heal insect bites. In addition, a study reported in 2015 by Brazilian researchers found that tea tree oil was effective against two fly species.

9. Dental Health

Although it is generally not recommended to take tea tree oil internally, it can be used for a variety of dental complaints. See “Precautions” below for more information about internal use.

In 2015, Indian researchers reviewed hundreds of studies on essential oils to investigate their therapeutic effect in a dentistry setting. They found that tea tree oil’s antibacterial and antifungal benefits made it a promising agent against oral pathogens.

Tea tree oil has also been found to be useful for periodontal disease. A 2016 Indian study found that a gel made from coenzyme Q10 and tea tree oil proved to be effective in the treatment of chronic periodontitis.

10. Skin Complaints
Psoriasis, eczema, dermatitis, sores, wounds, sunburn, warts, acne, and insect bites… down through the centuries all of these conditions have been eased using tea tree oil. Modern science is catching on to the reasons why it’s so beneficial.

A 2013 review of various studies on tea tree oil by Iranian scientists discusses its antimicrobial, anti-inflammatory and antioxidant properties. They also stated that tea tree oil accelerated wound healing and exhibited anti-skin cancer activity.

Note: To order the oil go to www.realoil.com and be sure to tell them I sent you!

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Microscopist And What He Found About Vinegar

 
MEDICAL SCIENCE:

The Lord opened the way for a close friend of mine to get in contact with a NUTRITIONAL MICROSCOPIST, who does Live Cell Analysis. His name is Jeffrey A. Arnson.

VINEGAR IS THE HIGHEST FORM OF FERMENTATION

Call the Microscopist to find out how harmful vinegar is

His email address is: jarnson@execpc.com

This man is not an SDA, but he shared the following with her: He takes the blood of humans and puts it under a microscope and magnifies it 20,000 times. He said you can see the red & white blood cells; bacteria; infection; how you eat; think; what you have done to your body within the last 90-120 days.

He said…when VINEGAR was added to the blood it did the following:

• Depletes WBC (white blood cell) count (this effects your IMMUNE SYSTEM)
• Can lead to anemia
• Robs nutrients from the body
• Thins out the blood
• Irritates the stomach
• Excites the blood (Remember that “The Life of the Flesh Is In The Blood” – Leviticus 17:11)

· The fermentation process of vinegar is converted into ACETIC ACID to become vinegar. ACETIC ACID COMES WITH AN MSDS SHEET.

http://msds.chem.ox.ac.uk/AC/acetic_acid.html

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NAC - (N-Acetylcysteine) and COVID-19

This is very important info; so I decided to post it. 

Hello Friends,

Many of you know that I have been at the forefront of controversy regarding the supplementation of NAC for Covid-19 after I posted an article on Facebook showing increased risk of adenocarcinoma with NAC use.  The following is a careful and very prayerful examination of this position. What I found was, in fact, much more of an issue than increased risk of cancer. Please read and provide feedback if you are able.

Regards,
Ron Meinhardt
founder@healthislife.org

Introduction:

Reports of the efficacy of the compound N-acetylcysteine in improving Covid-19 outcomes have been circulating around lately in health and wellness circles. When asked of my own personal opinion with NAC in supplement form, I replied that I had ceased taking or recommending its use because of the risk for cancer initiation often accompanying protein isolate or amino acid supplementation (1,2,3,4). However, since very recently being challenged on this hypothesis, I have dug deeper into the voluminous amount of research surrounding this substance. The purpose of this report is to share some of my findings. If you are currently taking this as a dietary supplement I strongly encourage you to read this article in its entirety.

NAC - What is it?

Here’s a little background info I found surprising… L-cysteine, the main ingredient in N-Acetylcysteine, has been historically manufactured in foreign countries (in particular China) from human hair (a potent source of the amino acid). Other sources may include bird feathers or even synthetic derivation (5). One of the more popular processes in which L-cysteine is then “acetylated" into NAC is described as follows: “we have prepared […NAC…] using 1 equiv. of acetic anhydride [which is basically vinegar without moisture] and a variety of acid acceptors in aqueous tetrahydrofuran)” (6). Furthermore, undesired byproducts (from 35-20%) will need to be removed although even higher purity varieties may contain some degree of contaminants (7).

Therapeutic effects:

There are many studies showing benefit in vitro (outside the body) and vivo (inside the body), of the indirect free radical scavenging effects of NAC (8). However as one researcher wrote: “NAC should not be considered to be a powerful antioxidant in its own right: its strength is the targeted replenishment of GSH [Glutathione] in deficient cells and it is likely to be ineffective in cells replete in GSH” (9). 

The role of NAC in the treatment many diseases is still unclear and controversial. It has been shown to possess strong mucolytic properties (by breaking the disulfide bonds in mucus) and is frequently given in cases of acetaminophen overdose to decrease liver toxicity (10). However, one article, reviewing over one hundred and three research studies related to the clinical use of this compound, found that “numerous, mainly small clinical trials with variable doses have yielded inconsistent results in a wide variety of diseases” (11). Furthermore, some of the reported benefits of NAC were shown to be due to dosages that would be difficult to achieve in the clinical setting, in particular with oral supplementation (12).

Unexpected side effects:

Besides potentially increasing the risk of cancer (1,2,3,4), NAC has been found to decrease the cancer-fighting effectiveness of a large class of naturally medicinal substances, such as curcumin (13), berberine (14), vitamin D (15), melatonin (16), selenium (17), and cucurbitacin (18). This is due to the fact that these compounds utilize oxidation (aka production of free radicals) to initiate cancer cell death (apoptosis) (13,14,15,16,17,18) and the robust and persistent repletion of glutathione by NAC neutralizes this effect.

This point deserves further elaboration due to the misconception that antioxidants are usually seen as “good” and free radicals “bad.” While persistently disarming free radicals (aka reactive oxygen species or R.O.S.) is a great idea in many disease processes it can be dangerous in others. In the case of pulmonary disease: “based on this knowledge, some researchers suggested that a faster clearance of ROS could reduce lung damage and improve lung function.” (19) However, “ROS generation by […] pathogens has also been established in respiratory epithelial cells, and the modulation of ROS was reported to be important for respiratory virus–induced innate immune mechanisms” (ibid).

An example of the positive effects of NAC’s action are the studies showing significant reduction of the severity of some influenza infections (20, 21, 22). This may be due largely to the fact that the virus is spread readily through replication (multiplication inside the cell) and consequent rupture of the cell membrane (apoptosis) (23). According to one study: “apoptosis induction is an antiviral host response, however, influenza A virus (IAV) infection promotes host cell death” (ibid). This allows the virus, after sufficient intercellular replication, to exit the cell in masse. Apoptosis is triggered by the overwork of the virus-hijacked cell’s organelles (mitochondria and endoplasmic reticulum) and production of large amounts of oxidative stress. NAC can suppress these free radicals and reverse the trend toward apoptosis and subsequent release of virons, thus slowing the spread of the Influenza virus.

While this process may be potentially beneficial in influenza infection, it may actually be dangerous in human coronavirus infection. One reason is that the coronavirus can efficiently exit the host cell the process of viral budding instead of apoptosis (24). In fact, cells infected with coronavirus can even adhere and fuse with neighboring cells “...lead[ing] to the formation of giant, multinucleated cells, which allows the virus to spread within an infected organism without being detected or neutralized by virus-specific antibodies” (Ibid). This can lead to asymptomatic infection but with massive amount of viral shedding, thus potentially making an infected person apparently healthy but extremely contagious. 

During the replication of coronaviruses "massive amount[s] of structural proteins [are] synthesized to assembly progeny virions. The production, folding, and modification of these proteins undoubtedly increase the workload of the ER [endoplasmic reticulum]” (24).. However, coronaviruses naturally block a critical pathway (the integrated stress response) that would normally trigger a decrease of viral replication and apoptosis of the infected cell. This is at least in part by limiting the secretion of the cell’s red alert compounds known as “interferons” (Ibid). However, interferon (alpha) production can still occur (leading to apoptosis) due to the massive amount of oxidative stress generated by viral production (Ibid). Thus, production of functional interferon after infection with SARS-CoV-1 is purported to be "essential for the control of potentially lethal coronavirus infections” (25, 26).

NAC and coronavirus replication:

Reducing the free radical generation by stressed organelles with N-acetylsysteine (a potent and persistent replenisher of intercellular glutathione) has been shown to potentially encourage an environment supporting enhanced viral replication in human coronavirus infection (resulting in enhanced endoplasmic reticulum folding capacity) (27). To make matters worse, NAC may alter the profile of the normal production of cytokines (chemical messengers mediating the inflammatory response) (19, 27).

The latter is concerning seeing that immune dysregulation is already a hallmark of coronavirus infection. A study in the journal Clinical Infectious Diseases “observed leukopenia in 47% of patients, lymphopenia in 84%, and T lymphopenia in 95%. CD4(+) T lymphocyte levels were reduced in 100% of patients, CD8(+) T lymphocyte levels were reduced in 87%, B lymphocyte levels were reduced in 76%, and natural killer cell levels were reduced in 55%” (28).

Also of interest, in HIV infection, dosages of NAC typical of oral administration, but not intravenous, caused an increase in the infection rate of monocytes (27). After viral infection these immune cells are themselves at increased risk for aberrant behavior, potentially letting go of their munitions in the lungs and causing a devastating condition known as cytokine storm- the single most deadly end result of Covid-19 infection (29, 30)

Where do we go from here?

Ultimately we will need to weigh the evidence ourselves and make an educated decision. The good news is that we can safely enjoy the benefits of dietary bioavailable cysteine and thereby ensure healthy normalization of glutathione levels. For example, raw garlic contains natural forms of this amino acid, such as S-allyl-cysteine, which also shows potent anti-cancer effects (31, 32, 33). Furthermore, adequate amounts of vitamin D have been shown to safely regulate levels of glutathione (34)

By now you may have developed second thoughts about utilizing N-acetylcysteine, or for that matter, any substance that has not been adequately tested, against Covid-19. What then can we rely upon in the face of such an unknown threat? 

Here is some timeless counsel:

“The simpler remedies are less harmful in proportion to their simplicity; but in very many cases these are used when not at all necessary. There are simple herbs and roots that every family may use for themselves and need not call a physician any sooner than they would call a lawyer.” — 2nd Selected Messages, page 279 

“When physicians understand physiology in its truest sense, their use of drugs will be very much less, and finally they will cease to use them at all. The physician who depends upon drug medication in his [practice] shows that he does not understand the delicate machinery of the human organism.”—Unpublished Testimonies, October 12, 1896.

“Educate away from drugs. Use them less and less, and depend more upon hygienic agencies; then nature will respond to God's physicians—pure air, pure water, proper exercise, a clear conscience.” — The Medical Ministry, page 259

“Pure air, sunlight, abstemiousness, rest, exercise, proper diet, the use of water, trust in divine power—these are the true remedies.”—The Ministry of Healing, page 127 

In summary:

The concept of temperance is shown to apply even at the molecular level: moderate use of that which is good, and complete abstinence of that which is harmful. Normal levels of intercellular glutathione have a positive effect upon our health, however, given the raw materials, the body can do a better job of managing the fine line of production so that the compound not used as a cloak for cancer or viral development. Let us all seek to become better acquainted with this principle, for we are entering upon an era in this world’s history that will necessitate more careful practices than heretofore have characterized our work.

References:

1) JCI Insight. 2019 Oct 3;4(19). pii: 127647. doi: 10.1172/jci.insight.127647.

2) Sci Transl Med. 2014 Jan 29;6(221):221ra15. doi: 10.1126/scitranslmed.3007653.

3) Rejuvenation Res. 2014 Jun;17(3):306-11. doi: 10.1089/rej.2014.1577.

4) Sci Transl Med. 2015 Oct 7;7(308):308re8. doi: 10.1126/scitranslmed.aad3740.

5)https://oukosher.org/blog/consumer-kosher/what-could-be-wrong-with-l-cysteine/

6) N-Acylation of Cysteine Tellis A. Martin, John R. Corrigan, and Coy W. Waller

The Journal of Organic Chemistry 1965 30 (8), 2839-2840 DOI: 10..1021/jo01019a509 

7) https://www..sigmaaldrich.com/catalog/product/sial/a7250?lang=en&region=US&utm_medium=cpc&utm_source=bing&utm_term=n%20acetyl%20cysteine&utm_campaign=SIAL%20Position%20Support%20Global%20(Bing%20ebizpfs)&utm_content=sial/a7250

8) Oxid Med Cell Longev. 2018; 2018: 2835787.

9) Pharmacol Ther. 2014 Feb;141(2):150-9. doi: 10.1016/j.pharmthera.2013.09.006. Epub 2013 Sep 28. 

10) Sci Transl Med. 2015 Feb 25; 7(276): 276ra27.  doi: 10.1126/scitranslmed.3010525  

11) Br J Clin Pharmacol. 2006 Jan; 61(1): 5–15.

12) J Cardiovasc Pharmacol. 2009 Oct;54(4):319-26. doi: 10.1097/FJC.0b013e3181b6e77b.

13) Korean J Physiol Pharmacol. 2010 Dec;14(6):391-7. doi: 10.4196/kjpp.2010.14.6.391. 

14) Int J Oncol. 2011 Feb;38(2):485-92. doi: 10.3892/ijo.2010.878. 

15) J Steroid Biochem Mol Biol. 2011 Jan;123(1-2):85-9. doi: 10.1016/j.jsbmb.2010.11.010.  

16) Basic Clin Pharmacol Toxicol. 2011 Jan;108(1):14-20.. doi: 10.1111/j.1742-7843.2010.00619.x.  

17) J Trace Elem Med Biol. 2003;17(1):19-26.  

18) Anticancer Agents Med Chem. 2014;14(8):1146-53.  

19) Am J Respir Cell Mol Biol. 2013 Nov; 49(5): 855–865..

20) Biochem Pharmacol. 2011 Sep 1;82(5):548-55. doi: 10..1016/j.bcp.2011.05.014. Epub 2011 May 25.    

21) J Negat Results Biomed. 2011 May 9;10:5. doi: 10.1186/1477-5751-10-5.  

22) Eur Respir J. 1997 Jul;10(7):1535-41.   

23) Cell Death Dis. 2013 Mar 28;4:e562. doi: 10.1038/cddis.2013.89.   

24) Front Microbiol. 2014; 5: 296.  Published online 2014 Jun 17. doi: 10.3389/fmicb.2014.00296. 

25) Blood. 2007 Feb 1;109(3):1131-7. Epub 2006 Sep 19.

26) J Virol. 2007 Aug; 81(16): 8692–8706..  

27) JCI Insight. 2016 Dec 8; 1(20): e88255. 

28) Clin Infect Dis. 2003 Sep 15;37(6):857-9. Epub 2003 Aug 28.. 

29) Intensive Care Med. 2020 Mar 3. doi: 10.1007/s00134-020-05991-x. [Epub ahead of print].

30) Semin Immunopathol. 2017; 39(5): 529–539.

31) Exp Ther Med. 2020 Feb;19(2):1511-1521. doi: 10.3892/etm.2019.8383.

32) Int J Mol Sci. 2020 Feb 6;21(3). pii: E1090. doi: 10.3390/ijms21031090.. 

33) Int Immunopharmacol. 2019 Apr;69:19-26. doi: 10.1016/j.intimp.2019.01.026. Epub 2019 Jan 18.  

34) J Neurochem. 1999 Aug;73(2):859-66.  

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PLEASE DONATE TODAY, IF the Holy Spirit impresses you, (donate button below and on the side) so I can continue to help others. Thank you! If you can't see the donate button or want quick access – use this link: PayPal.Me/ReneeB

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